Benign Prostatic Hyperplasia

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Benign Prostatic Hyperplasia

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Analysis of the risk factors for incidental carcinoma of the prostate in patients with benign prostatic hyperplasia. (Clinics. 2006) " CONCLUSIONS: Advanced age and the presence of a suspect digital rectal examination represent the most important risk factors for the diagnosis of an incidental carcinoma of the prostate. However, the low positive predictive values reflect the weak correlations among these variables."

BPH:Epidemiology and comorbidities. (Am J Manag Care. 2006) "Recently published data suggest that clinical benign prostatic hyperplasia (BPH), which is hallmarked by the occurrence of moderate-to-severe lower urinary tract symptoms (LUTS), occurs in about one quarter of men in their 50s, one third of men in their 60s, and about half of all men 80 years or older. Although effective treatments for LUTS/BPH are available, this condition often occurs in the context of common, age-related comorbidities such as cardiovascular disease, hypertension, and erectile dysfunction."

Could inflammation be a key component in the progression of benign prostatic hyperplasia? (Curr Opin Urol. 2006)

Effect of the consumption of tomato paste on plasma prostate-specific antigen levels in patients with benign prostate hyperplasia. (Braz J Med Biol Res. 2006) "Dietary ingestion of 50 g of tomato paste per day for 10 weeks significantly reduced mean plasma PSA levels in patients with benign prostate hyperplasia, probably as a result of the high amount of lycopene in tomato paste. This was not a prostate cancer prevention study, but showed some action of tomato paste in prostate biology. The development of prostate cancer is typically accompanied by an increase in plasma PSA levels, thus any intervention that affects plasma PSA levels can suggest an impact in the progression of disease."

Enlarged prostate: A landmark national survey of its prevalence and impact on US men and their partners (Prostate Cancer and Prostatic Diseases (2006))

How can we best characterize the relationship between erectile dysfunction and benign prostatic hyperplasia? (J Sex Med. 2006) "INTRODUCTION: Considerable epidemiological literature exists, showing a relationship between voiding dysfunction and sexual dysfunction. However, few basic science or prospective data are available to determine the nature of the relationship between benign prostatic hyperplasia (BPH)/lower urinary tract symptoms (LUTS) and sexual dysfunction. … CONCLUSION: At present, the epidemiological data do not support a causal relationship based on Hill's criteria for causality between LUTS and ED but suggest a less rigorous association. Additional research is needed to better characterize this relationship."

Is Benign Prostatic Hyperplasia (BPH) an Immune Inflammatory Disease? (Eur Urol. 2006) "CONCLUSIONS: These data strongly suggest that BPH is an immune inflammatory disease."

Oxidative stress and antioxidant status in non-metastatic prostate cancer and benign prostatic hyperplasia. (Clin Biochem. 2006)

Predictors of Recurrence in Bipolar Disorder: Primary Outcomes From the Systematic Treatment Enhancement Program for Bipolar Disorder (STEP-BD). (Am J Psychiatry. 2006)

The natural history of benign prostatic hyperplasia. (BJU Int. 2006) "Thus, there is evidence from longitudinal studies, and to a lesser extent from the placebo arms of large controlled studies, that BPH is a progressive disease. Symptom worsening is by far the most frequently occurring progression event. Identifying those patients at risk of BPH progression is crucial to optimize their management."

Tracking of longitudinal changes in measures of benign prostatic hyperplasia in a population based cohort. (J Urol. 2006)

Vascular resistance in the prostate evaluated by colour Doppler ultrasonography: is benign prostatic hyperplasia a vascular disease? (BJU Int. 2006) "The present results support the hypothesis that an age-related impairment of blood supply to the lower urinary tract might have a role in the development of BPH."

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