NeuroDegeneration
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NeuroDegeneration
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JOURNAL ARTICLES:
Brain ferritin iron may influence age- and gender-related risks of neurodegeneration. (Neurobiol Aging. 2006)
Genes and the Environment in Neurodegeneration. (Biosci Rep. 2006) "In this review we highlight some of the major neurodegenerative disorders (Alzheimer's disease, Parkinson's disease, Amyotrophic lateral sclerosis, Huntington's disease, and prion diseases.) and discuss possible links of gene-environment interplay including, where implicated, mitochondrial genes."
Mechanisms of aluminum-induced neurodegeneration in animals: Implications for Alzheimer's disease. (J Alzheimers Dis. 2006) "Rabbits are particularly sensitive to aluminum neurotoxicity and they develop severe neurological changes that are dependent on dose, age and route of administration. The most prominent feature induced by aluminum in rabbit brain is a neurofibrillary degeneration that shares some similarity with the neurofibrillary tangles found in Alzheimer's disease patients."
Neurodegeneration and neuroprotection in multiple sclerosis and other neurodegenerative diseases. ( Neuroimmunol. 2006)
Olfaction in neurodegenerative disorder. (Adv Otorhinolaryngol. 2006) "There has been gradual increase of interest in olfactory dysfunction since it was realised that anosmia was a common feature of idiopathic Parkinson's disease (IPD) and Alzheimer-type dementia. It is an intriguing observation that a premonitory sign of a disorder hitherto regarded as one of movement or cognition may be that of disturbed sense of smell. In this review of aging, IPD, parkinsonian syndromes, tremor, Alzheimer's disease (AD), motor neuron disease (MND), Huntington's chorea (HC) and inherited ataxia, the following observations are made: (1) olfactory senescence starts at about the age of 36 years in both sexes and accelerates with advancing years, involving pleasant odours preferentially; (2) olfactory dysfunction is near-universal, early and often severe in IPD and AD developing before any movement or cognitive disorder; (3) ."
The brain as a target of inflammation: common pathways link inflammatory and neurodegenerative diseases. (Trends Neurosci. 2006) "Classical knowledge distinguishes between inflammatory and non-inflammatory diseases of the brain. Either the immune system acts on the CNS and initiates a damage cascade, as in autoimmune (e.g. multiple sclerosis) and infectious conditions, or the primary insult is not inflammation but ischemia or degeneration, as in stroke and Alzheimer's disease, respectively. However, as we review here, recent advances have blurred this distinction. . "
The role of aluminum and copper on neuroinflammation and Alzheimer's disease. (J Alzheimers Dis. 2006)
The role of inflammation in CNS injury and disease. (Br J Pharmacol. 2006)
Thiamine deficiency induces endoplasmic reticulum stress in neurons. (Neuroscience. 2006)
Use of Ginseng in Medicine With Emphasis on Neurodegenerative Disorders. (J Pharmacol Sci. 2006)
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