Brain Injury
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Notes
The 2007 Treatment Guidelines section will contain the 2007 published guidelines. To view Guidelines from previous years, view year 2006 Treatment Guidelines and 2005 InfoMedlinks (Articles section) or our Monthly Online Newsletter (under the Guidelines section).
Brain Injury
Daily Treatment Report
Cognitive Therapy-CBT-Psychotherapy
Device Therapy
Drug Side-Effects and Interactions
Drugs
Post-traumatic seizure disorder following acquired brain injury (Brain Injury 2007) "Conclusions: Prophylactic anti-convulsants are effective in reducing seizures in the first week post-injury in adults. However, they do not reduce the occurrence of seizures after the first week."
Mannitol for acute traumatic brain injury. (Cochrane Database Syst Rev. 2007)
Exercise
General Information
Recovery slow after surgery for severe brain bleed "When people suffer a serious type of bleeding in the brain called a subarachnoid hemorrhage, caused by a ruptured blood vessel, surgery can sometimes stop further damage occurring. Even so, cognitive impairment is common and recovery can take months, researcher report."
Severe Traumatic Brain Injury in Austria VI: Effects of guideline-based management. (Wien Klin Wochenschr. 2007)
Severe Traumatic Brain Injury in Austria V: CT findings and surgical management. (Wien Klin Wochenschr. 2007)
Severe Traumatic Brain Injury in Austria IV: Intensive care management. (Wien Klin Wochenschr. 2007)
Concerns Raised Over Head Injury Studies "Using high-dose mannitol to treat head injuries may not be a sound strategy as the three main studies supporting this practice may not have even taken place, according to a report in British Medical Journal for February 24."
Traumatic brain injury-mediated hypopituitarism. Report of four cases. (Eur J Pediatr. 2007) " Growth hormone was the most commonly affected pituitary hormone. The time interval between the injury and diagnosis of pituitary hormone deficiency was between 2.5 weeks to 1.5 years. Hormone replacement therapy permitted normal completion of growth and development. Awareness among physicians treating children with traumatic brain injury of the risk of hypopituitarism is necessary to optimise the outcome."
Guidelines
Immunotherapy
Internet Sites
Treatment Information
Drug-Food-Supplement Information
DrugDigest (drug interactions)
FDA - Drug Interactions: What You Should Know
NIH - Botanical Dietary Supplements: Background Information
NIH - Drug, Supplements, and Herbal Information
NIH - Herbal Supplements: Consider Safety, Too
NIH - Vitamin and Mineral Supplement Fact Sheets
Nutrition
Other
Other Treatments
Cooling the injured brain: how does moderate hypothermia influence the pathophysiology of traumatic brain injury. (Curr Pharm Des. 2007)
Radiotherapy
Supplements-Vitamins-CAM
Omega-3 Fatty Acids Supplementation Restores Mechanisms that Maintain Brain Homeostasis in Traumatic Brain Injury. (J Neurotrauma. 2007) "Our results suggest that TBI may compromise neuronal protective mechanisms by involving the action of Sir2alpha. In addition, results show the capacity of omega-3 fatty acids to counteract some of the effects of TBI by normalizing levels of molecular systems associated with energy homeostasis."
DHEAS repeated treatment improves cognitive and behavioral deficits after mild traumatic brain injury. (Eur Neuropsychopharmacol. 2007)
The potential of melatonin in reducing morbidity-mortality after craniocerebral trauma. (J Pineal Res. 2007) " Some of the actions of melatonin that support its pharmacological use after CCT include its role as a scavenger of both oxygen and nitrogen-based reactants, stimulation of the activities of a variety of antioxidative enzymes (e.g. superoxide dismutase, glutathione peroxidase, glutathione reductase and catalase), inhibition of pro-inflammatory cytokines and activation-adhesion molecules which consequently reduces lymphocytopenia and infections by opportunistic organisms. The chronobiotic capacity of melatonin may also reset the natural circadian rhythm of sleep and wakefulness. Melatonin reduces the toxicity of the drugs used in the treatment of CCT and increases their efficacy. Finally, melatonin crosses the blood-brain barrier and reduces contusion volume and stabilizes cellular membranes preventing vasospasm and apoptosis of endothelial cells that occurs as a result of CCT."
Surgery
The role of surgery in traumatic brain injury. (Curr Opin Crit Care. 2007) " SUMMARY: A surgical approach is frequent in posttraumatic intracranial hematomas in spite of a low level of evidence. One of the surgical options either in association with hematoma evacuation or in isolation is the technique of bone flap decompression."
Transplantation
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