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Vitamins

Vitamin E

Vitamin E may reduce blood clots in women: study

NEWS:

ARTICLES:

JOURNAL ARTICLES:

Anti-inflammatory properties of alpha- and gamma-tocopherol. (Mol Aspects Med. 2007)

Effects of Random Allocation to Vitamin E Supplementation on the Occurrence of Venous Thromboembolism. Report From the Women's Health Study (Circulation 2007) "Conclusions—These data suggest that supplementation with vitamin E may reduce the risk of VTE in women, and those with a prior history or genetic predisposition may particularly benefit."

Ozone enhancement of lower airway allergic inflammation is prevented by gamma-tocopherol. (Free Radic Biol Med. 2007)

The role of vitamin E in the prevention of cancer: a meta-analysis of randomized controlled trials. (Ann Saudi Med. 2007) “CONCLUSIONS: Vitamin E supplementation was not associated with a reduction in total mortality, cancer incidence, or cancer mortality, but it was associated with a statistically significant reduction in the incidence of prostate cancer. Vitamin E can be used in the prevention of prostate cancer in men who are at high risk of prostate cancer.”

Tocotrienols: the emerging face of natural vitamin e. (Vitam Horm. 2007) "Natural vitamin E includes eight chemically distinct molecules: alpha-, beta-, gamma-, and delta-tocopherols and alpha-, beta-, gamma-, and delta-tocotrienols. More than 95% of all studies on vitamin E are directed toward the specific study of alpha-tocopherol. The other forms of natural vitamin E remain poorly understood. … Current developments in vitamin E research clearly indicate that members of the vitamin E family are not redundant with respect to their biological functions. alpha-Tocotrienol, gamma-tocopherol, and delta-tocotrienol have emerged as vitamin E molecules with functions in health and disease that are clearly distinct from that of alpha-tocopherol. … A rapidly expanding body of evidence supports that members of the vitamin E family are functionally unique. In recognition of this fact, title claims in publications should be limited to the specific form of vitamin E studied. For example, evidence for toxicity of a specific form of tocopherol in excess may not be used to conclude that high-dosage "vitamin E" supplementation may increase all-cause mortality. Such conclusion incorrectly implies that tocotrienols are toxic as well under conditions where tocotrienols were not even considered."

Vitamin E and selenium supplementation and risk of prostate cancer in the Vitamins and lifestyle (VITAL) study cohort. (Cancer Causes Control. 2007) "CONCLUSIONS: In this prospective cohort, long-term supplemental intake of vitamin E and selenium were not associated with prostate cancer risk overall; however, risk of clinically relevant advanced disease was reduced with greater long-term vitamin E supplementation."

Vitamin E dietary supplementation significantly affects multiple risk factors for cardiovascular disease in baboons. (Am J Clin Nutr. 2007) "These effects have contrasting associations with CVD risk and may help account for the mixed results from clinical trials of dietary vitamin E."

Vitamin e during pre- and postnatal periods. (Vitam Horm. 2007)

Vitamin E, antioxidant and nothing more. (Free Radic Biol Med. 2007)

INTERNET SITES:

NIH - Medical Encyclopedia Vitamin E

NIH - Medical Encyclopedia Vitamin E source

NIH - Vitamin E

NIH - Vitamin E (Systemic)

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